Posts tagged #hand

Hand Nerve Blocks

Written by: Aldo Gonzalez, MD (NUEM ‘23) Edited by: Jason Chodakowski, MD (NUEM '20)
Expert Commentary by: Mike Macias, MD


Hand Nerve Blocks

Nerve blocks are the use of anesthetics to anesthetize an area by injecting directly around the nerve that innervates a certain area. It is useful when there is a large area to provide anesthesia, the area might get distorted by local infiltration and make it difficult to close the tissue, or the distribution of the area to be anesthetized is well-suited to a nerve block.

Indications

Nerve blocks of the median, ulnar, radial, and digital nerves are useful for injuries of the hand including fractures, lacerations, and burns.

Contraindication

  • Overlying infection

  • Previous allergic reaction to anesthetic

 Anesthetics

Landmark versus Ultrasound guidance

Ultrasound guidance is preferred given the ability to visualize the desired nerve and proper instillation of the anesthetic around the nerve. On ultrasound nerves are circular or triangular hyperechoic structures with hypoechoic structures within. Often described as having a “honeycomb” appearance as seen in the image of the median nerve below.

Materials

  • Ultrasound with Linear Transducer

  • Probe Cover

  • Sterile Ultrasound Gel

  • Anesthetic

  • 10 cc syringe

  • 18 gauge needle (to draw medication)

  • 25-27 gauge needle at least 1.5 in in length

  • Antiseptic Solution (ex. Chlorhexidine)

  • Towel

Positioning

The patient can be either supine or seated with their arm slightly abducted and rested on a flat surface. Their elbow can be flexed with the wrist supinated and in slight extension. A rolled towel can be used for patient comfort and help in maintaining slight extension.

Figure 1: Nerves, arteries, and muscles of the human forearm

Radial Nerve Block

The superficial radial nerve travels between the flexor carpi radialis and the radial artery on anterior (volar) and lateral (radial) aspect of the forearm. Near the wrist the radial nerve splits into the medial and lateral branch of the superficial radial nerve. The block of this nerve should be performed at the mid-forearm to distal third of the forearm before the nerve splits. The nerve may be difficult to see at the distal forearm so instead it can be found proximally and followed distally. A lateral (radial) approach of the forearm provides the most direct route to the nerve.

Figure 2: Demonstration of a radial nerve block using a lateral (radial) approach with in-plane ultrasound technique on a patient’s right hand. Radial nerve (yellow line) and ultrasound probe location (blue line).

Ulnar Nerve Block

At the distal forearm the ulnar nerve runs on the medial (ulnar) and anterior (volar) aspect of the forearm between the flexor carpi ulnaris tendon and the ulnar artery. The ulnar nerve lies in very close proximity to the ulnar artery in the distal forearm and increases the risk of accidental intravascular injection. It is safer to identify the ulnar nerve distally and the follow the artery and nerve proximally. Around the proximal third of the forearm the ulnar artery dives deeper and separates from the ulnar nerve. This provides a safer target. A medial (ulnar) approach of the forearm provides the most direct route to the nerve.

Figure 3: Demonstration of an ulnar nerve block using a median (radial) approach with in-plane ultrasound technique on a patient’s right hand. Ulnar nerve (yellow line) and ultrasound probe location (blue line).

Median Nerve Block

At the mid to distal forearm the median nerve runs in the middle of the anterior (volar) aspect of the forearm between the flexor digitorum superficialis and flexor digitorum profundus muscles/tendons. Near the wrist the nerve can be difficult to appreciate due to all the tendons of the anterior compartment of the arm. The nerve can be best appreciated at the mid-forearm. A lateral (radial) or medial (ulnar) approach can be used for in-plane technique or a mid-line approach using out-of-plane technique. Be mindful to avoid accidentally puncturing the radial or ulnar artery If using an in-plane technique with a lateral or medial approach.

Figure 4: Demonstration of a median nerve block using a midline approach with out-of-plane ultrasound technique on a patient’s right hand. Median nerve (yellow line) and ultrasound probe location (blue line).

Figure 5: Demonstration of a median nerve block using a median (ulnar) approach with in-plane ultrasound technique on a patient’s right hand. Median nerve (yellow line) and ultrasound probe location (blue line).

Figure 6: Demonstration of a median nerve block using a lateral (radial) approach with in-plane ultrasound technique on a patient’s right hand. Median nerve (yellow line) and ultrasound probe location (blue line).

Steps for Ultrasound-Guided Nerve Block

  1. Document a neurological exam prior starting the procedure

  2. Select the nerve or nerves best suited to achieve best anesthesia for the injury

  3. Use the linear transducer to visualize the nerve prior beginning the procedure

  4. Plan an approach and select the best site

  5. Draw up anesthetic in the 10 cc syringe with an 18 G needle

  6. Replace 18 G needle with 25-27 G needle

  7. Use antiseptic solution to prepare the skin

  8. Dawn sterile gloves

  9. Cover transducer in sterile cover

  10. Use ultrasound to visualize the nerve and confirm approach

  11. Insert the needle into the skin

  12. Advance the needle using in-plane or out-of-plane technique 

  13. Come close to the nerve but do not puncture the nerve

  14. Draw back to confirm not with-in a vessel

  15. Deliver 5mL of anesthetic

  16. The nerve will become enveloped in hypoechoic anesthetic and peel away from the fascia of nearby muscles

  17. Withdraw the needle.

  18. Wait 3-5 minutes until patient is fully anesthetized

References

  1. Drake, R., Vogl, A. W., & Mitchell, A. W. (2015). Gray's Anatomy for Students (3rd ed.): Elsevier.

  2. Farag, E., Mounir-Soliman, L., & Brown, D. L. (2017). Brown’s Atlas of Regional Anesthesia (5th ed.): Elsevier.

  3. Gray, H. (2000). Gray's Anatomy of the Human Body. 20th edition. Retrieved from https://www.bartleby.com/107/

  4. Harmon, D., Barrett, J., Loughnane, F., Finucane, B. T., & Shorten, G. (2010). Peripheral Nerve Blocks and Peri-Operative Pain Relief (2nd ed.): Elsevier.

  5. Pester, J. M., & Varacallo, M. (2019). Ulnar Nerve Block Techniques. In StatPearls [Internet]: StatPearls Publishing.

  6. Roberts, J. R., Custalow, C. B., & Thomsen, T. W. (2019). Roberts and Hedges' clinical procedures in emergency medicine and acute care (7th ed.): Elsevier.

  7. Waldman, S. D. (2016). Atlas of Pain Management Injection Techniques E-Book (4th ed.): Elsevier.

  8. Waldman, S. D. (2021). Atlas of Interventional Pain Management E-Book (5th ed.): Elsevier.


Expert Commentary

Thank you Drs. Gonzalez and Chodakowski for the excellent post on forearm nerve blocks! This is an important skill that definitely improves the care of our patients, especially since hand injuries are such a common emergency department presentation. This is especially true for injuries that are difficult to anesthetize using traditional local injection such as dog bites, burns, abscesses, large lacerations, and fractures of the hand. I’d like to dive a little deeper into a few aspects of forearm nerve blocks:

Ultrasound guidance

I think that the days of a landmark based approach to the majority of nerve blocks are gone with the widespread availability of ultrasound and its superiority with respect to block success and reduced complications. So if you have it, use it! 

Which nerve to block?

Once you have made the commitment to block one forearm nerve, it doesn’t require much additional time or effort to block a second or even a third! Often, hand injuries will span several nerve distributions so make sure you are providing adequate anesthesia. Here is a quick way to think of it: 

  • Major hand injury (ie burn, multiple hand fractures): Triple block 

  • Injury to radial aspect of hand or digits 1-4: Radial + median nerve block 

  • Injury to ulnar aspect of hand or 5th digit (ie Boxer’s fracture): Ulnar nerve block

It is important to remember that forearm nerve blocks do not provide anesthesia to the volar forearm or wrist and therefore will not be adequate for distal radius fracture reduction. In this case, an above the elbow Radial nerve block should be performed. 

Which local anesthetic should I use?

It’s always important to consider what the goals of your local anesthetic are when determining which one to use. If you are performing a quick procedure, the shorter the better such as lidocaine. If you are providing prolonged pain management such as with a burn, bupivicaine is a better choice. I tend to prefer lidocaine + epinephrine (duration of acton 2-2.5 hours) for most of my hand injuries. Why? In a busy emergency department managing many patients at a time, the initial block and the procedure you plan on performing (ie lac repair, fracture reduction, etc) do not always happen simultaneously (ie patient may still need x-ray, irrigation, ring removal, etc). Using lidocaine + epinephrine will allow you to provide immediate pain relief for your patient but give you time to do other tasks before the patient is ready for the procedure

Positioning

As with any procedure, the set up is extremely important. You nicely described positioning earlier but I just want to highlight a couple additional points. Make sure your patient is comfortable and your ultrasound screen is in-line with your procedure. You don’t want to be turning your head away from your block to look at the screen. For the median and radial nerve block, the patient’s arm should be supinated and resting on a hard flat surface. Both nerves can then be approached using an in-plane technique from the radial aspect of the arm. The ulnar nerve can be cumbersome to get to with this same patient positioning so I recommend abducting the shoulder to about 90 degrees and placing the arm on a Mayo stand next to the patient. This will allow an in-plane approach from the ulnar aspect of the arm. I have also found this positioning technique helpful for the ulnar nerve block. 

Procedural Tips

I wanted to end with a couple of important procedural pearls I have learned during my experience with performing these blocks:

  • Perform a pre-block exam! Always make sure to perform and document a full neurological exam of the hand before you block any nerve. This is important because you want to make sure you know if any sensory or motor changes are present before your perform the block otherwise if a neurological deficit is noted after, it makes it difficult to tell if the block caused the new symptom (you can always wait until the anesthetic wears off but it may be awhile if you used bupivicaine). 

  • Follow the arteries! Sometimes it can be tricky to find the ulnar and radial nerves. The easiest method is to always start distally at the wrist. Both the radial and ulnar nerves run with their paired artery so if you start here and slide proximally, you should see the nerve split away from the artery around the mid forearm. Block them here! 

  • Target the fascial plane! The key to an effective forearm nerve block is “bathing” the nerve in anesthetic. You will want to see spread of the anesthetic around the nerve in a crescent shape, full circumferential spread is not needed. Since these nerves run in the fascial plane the goal is to get your needle tip into this plane to deposit anesthetic. There is never a need to actually touch the nerve so avoid this by aiming for the fascia and not the nerve. 

  • Protect the hand! After you perform a forearm nerve block be sure to communicate with nursing, consultants, and the patient regarding what block was performed and how long the effects will last. If a long acting agent was used such as bupivicaine, the hand should be splinted or arm placed in a sling and instructions provided to patient regarding care at home if they are being discharged. 


Thank you again for providing this excellent piece on forearm nerve blocks. I cannot stress enough how essential I think these blocks are to the toolkit of the modern emergency physician. I promise you once you add these to your practice your patient’s will thank you!

Michael Macias, MD

Global Ultrasound Director, Emergent Medical Associates 

Clinical Ultrasound Director, SoCal MEC Residency Programs



How To Cite This Post:

[Peer-Reviewed, Web Publication] Gonzalez, A. Chodakowski, J. (2021, Nov 29). Hand Nerve Blocks. [NUEM Blog. Expert Commentary by Macias, M]. Retrieved from http://www.nuemblog.com/blog/hand-nerve-blocks


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Posted on November 29, 2021 and filed under Procedures.

Flexor Tenosynovitis

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Written by: Kevin Dyer, MD (NUEM PGY-3) Edited by: Adnan Hussain (NUEM Alum ‘17) Expert commentary by: Aviram Giladi, MD


Case Presentation

A 29 year old right-handed male with no significant past medical history presents to the ED with left hand pain for the past 4 days. He reports that the pain started in the MCP joint of his left 2nd digit and was just “achy” at first, a mild 3/10. He noted associated swelling and erythema over the joint as well. Symptoms slowly became worse and he went to an urgent care facility yesterday where he was diagnosed with gout and sent home with NSAIDs and Norco. Overnight the pain became markedly worse, 8/10, and he began having subjective fevers. He denied any trauma to the area, no history of gout, no immune compromising diseases or medications, and no IV drug use.  In the ED his vitals were stable and he was afebrile. He appeared uncomfortable. His left 2nd digit had fusiform swelling, pain with passive extension, tenderness to percussion along the flexor sheath, and was held in slight flexion at rest. The MCP was noted to be erythematous with the erythema extending to the palmar surface of the hand.

Background

This patient’s exam was concerning for flexor tenosynovitis (FTS), an infection of the flexor tendon and its synovial sheath that can result in deformity, tendon necrosis and adhesions leading to loss of function, or loss of limb, especially if treatment is delayed [1]. The flexor tendon sheath consists of visceral and parietal layers and functions to provide a gliding surface and nutrition to the extrinsic tendons of the digits. Once bacteria are inoculated into the space between the two layers, the synovial fluid becomes a medium for bacterial growth and the closed nature of the sheath limits the host’s immune response to fight infection [2].

Most patients with FTS will endorse a traumatic injury occurring 2-5 days prior to ED presentation [2]. Pang et al noted that 57 of their 75 patients (76%) with flexor tenosynovitis were caused by a traumatic event [4]. Of these, 81% were caused by a puncture wound. Often times, the inciting injury may have been trivial and patients may not endorse an event. Therefore, it is incredibly important to still consider the diagnosis of FTS even if a traumatic component is missing from the patient’s history.

Patients should be asked about associated symptoms such as fever, chills, anorexia, and malaise. Additionally, questions assessing the patient’s handedness, immune status, proximal extent of the pain, and other sites of pain should also be asked.

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Microbiology

The most likely causative bacteria for FTS are skin flora. A review of four studies published within the past 10 years showed that out of 201 cases, 92 (46%) were caused by Staphylococcus with 20 (10%) being methicillin-resistant Staphylococcus aureus(MRSA). Streptococcus species were the cause of 30 cases (15%), gram negative bacteria were the cause of 28 cases (14%), and 18 (9%) were olymicrobial. Interestingly, 49 (24%) cases of confirmed FTS were culture negative, which has been attributed to early use of intravenous antibiotics or an aggressive immune response [4].

Diagnosis

The clinical diagnosis of FTS is based on the work of Dr. Allen B. Kanavel who described the four cardinal signs as:

  1. Fusiform swelling.

  2. Pain with passive extension of the digit.

  3. Tenderness over the flexor sheath.

  4. The digit held in slight flexion at rest.

No published study has validated the sensitivity and specificity of Kanavel’s signs, nor has there been a study validating inter-observer reliability. However, several studies have looked at the presence of the individual signs in patients diagnosed with the condition. Studies published by Pang et al and Nikkah et al had a combined 91 patients [3,6]. The most common sign was fusiform swelling and was present in 89 of 91 patients (98%). The second most common was pain on passive extension (73%), followed by tenderness over the flexor sheath (67%), and finger held in slight flexion (67%). Dailiana et al reported that only 54% of their 41 patients exhibited all four of Kanavel’s signs [5]. However, all of their patients displayed tenderness over the flexor sheath, which has been described by several authors as the most important sign when distinguishing FTS from other infections of the hand [4,5,8,9].

Image from EM in 5, used with permission from Anna Pickens, MD (10).

Image from EM in 5, used with permission from Anna Pickens, MD (10).

Work up for patients with suspected FTS should include plain films to rule out a retained foreign body and fractures [2]. Laboratory studies should include white blood count (WBC), C-reactive protein (CRP), and erythrocyte sedimentation rate (ESR). Bishop et al studied 71 patients with clinically diagnosed FTS, 69 of which were confirmed by operative findings or positive intraoperative cultures [7]. All 69 patients had elevation of at least one of the three inflammatory markers, a positive predictive value of 100%. They reported negative predictive values for WBC, ESR and CRP as 4%, 3%, and 13%, respectively. The two patients without FTS were diagnosed with calcific tendinitis, both patients had normal inflammatory markers. These results suggest that a positive inflammatory marker when FTS is suspected makes the likelihood of infection extremely high. However, normal inflammatory markers cannot reliably rule out an infection.

                 

Treatment

The two cornerstones for treating FTS are prompt administration of IV antibiotics and emergent hand surgery consultation. Antibiotic treatment should be guided by local antibiotic susceptibilities as well as the mechanism of infection. As discussed above, Staphylococcus, including MRSA, and Streptococcus species account for 61% of FTS cases whereas an additional 23% of cases are caused by gram negatives or are polymicrobial.  Therefore, broad spectrum antibiotics are required. A common approach is to use vancomycin with piperacillin/tazobactam [2]. Consultation with an infectious disease specialist or a clinical pharmacist should be considered for patients with antibiotic allergies, immunocompromise, or chronic infections.

 

Learning Points

  • FTS can cause a loss of hand function or a loss of limb if treatment is delayed.

  • Patients may not endorse a traumatic event and the diagnosis must still be considered despite this.

  • The clinical diagnosis of FTS is made using Kanavel’s Signs:

    • Fusiform swelling.

    • Pain with passive extension of the digit.

    • Tenderness over the flexor sheath.

    • The digit held in slight flexion at rest.

  • Staphylococcus and Streptococcus species account for 61% of infections.

  • The two cornerstones of treatment are:

    • Broad antibiotics – Vancomycin and Zosyn is sufficient

    • Emergent Hand Surgery Consultation


Expert Commentary

Thank you for putting together this nice case report.  Hand infections are common, and the challenge for the emergency provider is in deciding which patients are appropriate for a surgical consult (and for the surgery team, deciding which patients need surgery).  In the era of strong IV antibiotics, the timing and indications for intervention are shifting.  With early intervention using strict extremity elevation (hanging from the ceiling if possible) and IV antibiotics, we are avoiding surgery for some patients.  This includes some with early FTS, before all of the Kanaval ’cardinal signs’ are evident.  IV antibiotics have made it so that some patients with FTS, a condition traditionally considered to require surgery, are able to avoid surgery altogether.  Maintaining a high index of suspicion for these problems is important in seizing these opportunities. 

When specifically thinking about FTS, the notable challenge is that most patients (around 50% or more, as highlighted in the Dailiana article review) will present with only one or two ‘cardinal’ findings.  Identifying any potential inciting event – whether small puncture, cut, or even working in the garden – helps to increase your index of suspicion.  As the case highlights, many patients have a red joint, hand pain, or other presenting complaints that muddy the picture.  The obvious FTS patients are relatively easy to identify, but the other 50% or more can be very challenging to diagnose.

Many infection, gout, arthritis flare, “hand that’s swollen and red”, etc. patients have such pain that a good exam is difficult.  But, deciding on one of the four types of hand infection surgical emergencies – abscess, septic arthritis, purulent FTS, or necrotizing fasciitis – is critical.  If the patient will not tolerate passive extension of the finger, my preferred way to evaluate for FTS without being unnecessary cruel is by manually compressing the tendons in the distal 1/3 of the volar forearm.  You can try this on yourself – let your arm relax and then squeeze your forearm at the junction between the middle and distal 1/3 (where flexor tendons start to become distinct from muscles) and you can make your fingers flex; relax on the forearm and they will return to resting posture.  If that maneuver creates focal pain in the swollen finger, my concern for FTS goes up. 

Overall, high index of suspicion is critical.  Rule FTS out, not in – convince yourself the patient doesn’t have a potential surgical problem by doing whatever evaluation and early treatment you think is appropriate and following the course, rather than delaying intervention until the presentation is more obvious.  And, whenever in doubt, keep the patient NPO and consult a specialist so that a treatment plan can be put together without unnecessary delay or risk.   

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Aviram Giladi, MD, MS

The Curtis National Hand Center, MedStar Union Memorial Hospital


How to Cite this Post

[Peer-Reviewed, Web Publication] Dyer K, Hussain A (2019, January 28). Flexor Tenosynovitis [NUEM Blog. Expert Commentary by Giladi A]. Retrieved from http://www.nuemblog.com/blog/flexor-tenosynovitis.


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References

  1. Kennedy CD, Huang JI, Hanel DP. In brief: Kanavel’s signs and pyogenic flexor tenosynovitis.ClinOrthopRelat Res 2016;474:280–4.

  2. Hyatt MT, Bagg MR. Flexor Tenosynovitis.OrthopClin N Am 2017;48:217-27

  3. Pang HN, Teoh LC, Yam AK, et al. Factors affecting the prognosis of pyogenic flexor tenosynovitis. J Bone Joint Surg Am 2007; 89:1742.

  4. Draeger RW, Bynum DK Jr. Flexor tendon sheath infections of the hand. J Am AcadOrthopSurg 2012;20:373–82.

  5. Dailiana ZH, Rigopoulos N, Varitimidis S, et al. Purulent flexor tenosynovitis: factors influencing the functional outcome. J Hand SurgEurVol 2008;33:280–5.

  6. Nikkhah D, Rodrigues J, Osman K, Dejager L. Pyogenic flexor tenosynovitis: one year’s experience at a UK hand unit and a review of the current literature. Hand Surg 2012; 17:199.

  7. Bishop GB, Born T, Kakar S, et al. The diagnostic accuracy of inflammatory blood markers for purulent flexor tenosynovitis. J Hand Surg Am 2013;38:2208–11.

  8. Boles SD, Schmidt CC. Pyogenic flexor tenosynovitis.Hand Clin 1998;14:567–78.

  9. Pollen AG. Acute infection of the tendon sheaths. Hand 1974;6:21–5.

  10. Pickens, Anna. "Flexor Tenosynovitis." EM in 5. N.p., 20 Apr. 2014. Web. 10 May 2017.

 

Posted on January 28, 2019 and filed under Orthopedics.